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血管造影后发生的急性脑缺血发作(TIAs)可能为导管尖栓子形成进入颅内循环或导管引起动脉粥样硬化斑块脱落所致,常突然发生于造影台上,但晚发的患者少见。作者报导3例女性患者分别于主动脉弓造影、心导管检查和脑血管造影后6～48小时发生反复的TIAs症状,但14～20个月后随访无复发。显然,晚发的TIAs与血管造影有关,但其机制不明。为了证实血管造影和血小板活性的关系,作者测定了16例阻塞性脑血管病患者作为血小板活性指标的血浆β-血栓球蛋白(βTG)水平。分为两组:
Acute cerebral ischemic attacks (TIAs) after angiography may be caused by the formation of catheter emboli into the intracranial circulation or catheter-induced atherosclerotic plaque detachment, often occurs suddenly in the imaging stage, but late-onset patients Rare. The authors reported repeated TIAs in 6 women at 6 to 48 hours after aortic arch angiography, cardiac catheterization and cerebrovascular angiography, respectively, but no recurrence was observed at 14-20 months. Obviously, late-onset TIAs are associated with angiography, but the mechanism is unknown. To confirm the relationship between angiography and platelet activity, the authors measured plasma β-thromboglobulin (βTG) levels in 16 patients with obstructive cerebrovascular disease as an index of platelet activity. Divided into two groups: