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前言 1946年Watson与Hoffbuer报告8例,在肝炎小流行区临床生化与梗阻性黄疸相似,但无肝外胆道梗阻,肝活检见肝细胞变化轻微或无,小叶中心部淤胆,细胆管周围炎细胞浸润,推测是肝炎病毒引起,称为“细胆管炎性肝炎”。其后研究证明细胆管周围炎病变与胆汁淤滞程度无一定相关性,甚而无细胆管周围炎症细胞浸润。故这一命名并不确切。1960年Du—bin对急性病毒性肝炎肝活检病例分析结
Preface 1946 Watson and Hoffbuer reported 8 cases of clinical biochemistry and obstructive jaundice in the endemic areas of hepatitis is similar, but no extrahepatic biliary obstruction, liver biopsy showed little or no changes in liver cells, centrilobular cholestasis, peribiliary bile duct inflammation Cell infiltration, presumably caused by hepatitis virus, known as “cholangitis.” Subsequent studies have shown no correlation between bile duct inflammation and cholestasis, even without perivascular inflammatory cell infiltration. Therefore, this name is not exact. 1960 Du-bin analysis of liver biopsy cases of acute viral hepatitis