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为探讨海带多糖对超重环境下微波辐射大鼠氧化损伤的影响,将18只雄性Wistar大鼠随机分为3组,每组6只,制作大鼠6 g超重环境下微波(200 mW/cm2,5min)损伤模型(模型组),并灌胃给予20 mg/ml海带多糖(海带多糖组)。结果显示,模型组大鼠血清一氧化氮(NO)、抗超氧阴离子活力、过氧化氢酶(CAT)、一氧化氮合酶(NOS)、还原型谷胱甘肽(GSH)、谷胱甘肽过氧化物酶(GSH-Px)低于对照组,海带多糖组大鼠上述指标高于模型组;模型组大鼠肝匀浆中CAT、GSH-Px和GSH低于对照组,NOS高于对照组;海带多糖组大鼠肝匀浆中CAT高于模型组,NOS低于模型组,上述差异均有统计学意义(P<0.05)。提示海带多糖对超重环境下微波损伤的抑制作用可能与CAT、GSH-Px、GSH升高有关。
To investigate the effect of laminarin on the oxidative damage induced by microwave radiation in overweight environment, 18 male Wistar rats were randomly divided into 3 groups (6 rats in each group) 5min) injury model (model group), and intragastric administration of 20 mg / ml kelp polysaccharide (kelp polysaccharide group). The results showed that the levels of NO, SOD, CAT, NOS, GSH, The levels of GSH-Px and GSH-Px in the model group were lower than those in the control group. The levels of GSH-Px and GSH in liver homogenate of the model group were lower than those in the control group In the control group, CAT in the liver homogenate of the kelp polysaccharide group was higher than that of the model group, and the NOS was lower than that of the model group. The above differences were statistically significant (P <0.05). These results suggest that the inhibitory effect of kelp polysaccharides on microwave injury in overweight environment may be related to the increase of CAT, GSH-Px and GSH.