论文部分内容阅读
虽然已知人类色素膜炎有各种异常现象,但它们在疾病的病理生理学上是否重要,或系继发于非免疫组织损坏后的表面现象,则不清楚。循环的免疫复合物能在色素膜炎的实验模型中造成眼组织的破坏;视网膜血管炎及Fuchs异色性睫状体炎患者血清中也证实有免疫复合物,但其性质及其与疾病状态的关系仍不明确。作者对1977—1978年间对未发现金身病的慢性弥漫性色素膜炎14例(9—73岁,男5,女9)慢性虹膜睫状体炎16例(5—70岁,男4,女12)
Although there are various anomalies of human uveitis, it is unclear whether they are important in the pathophysiology of the disease or secondary to surface phenomena after non-immune tissue damage. Cyclic immune complexes can cause ocular tissue destruction in experimental models of pigmented inflammation; retinal vasculitis and Fuchs heterochromatic ciliary body inflammation in patients also confirmed immune complexes, but its nature and its relationship to disease status The relationship is still not clear. From 1977 to 1978, 14 patients (9-73 years old, 5 men and 9 women) with chronic diffuse uveitis who did not find gold body disease were enrolled in this study. 16 cases (5-70 years old, male 4, Female 12)