31例晕痣患者中伴发白癜风14例(45.1%),516例白癜风患者伴晕痣10例(1.9%)。晕痣痣细胞周围有密集的淋巴细胞浸润。痣细胞受损,最后消失。晕痣的皮肤内郎格罕细胞增多。晕部表皮中黑素细胞消失。提示晕痣的组织学特征符合免疫学改变。此外,还观察到晕痣组织中一种较为特殊的细胞,推测可能是郎格罕细胞在皮肤内行有丝分裂。 Fourteen cases (45.1%) were accompanied by vitiligo in 31 cases of halo nevus and 10 cases (1.9%) in 516 cases of vitiligo with halo nevus. Halo nevus cells around the dense lymphocytic infiltration. Mole cell damage, and finally disappeared. Halo nevus in the skin increased Langerhans cells. Halo melanocytes disappear in the halo part of the epidermis. Hint suggestive of Histological features consistent with immunological changes. In addition, a more specific cell in halo nevus was also observed, presumably due to mitosis of Langerhans cells in the skin.