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作者复制犬失血性休克模型。用改良的硫代巴比妥酸法测定血浆丙二醛（ＭＤＡ）含量，用光化学扩增法测定全血超氧歧化酶（ＳＯＤ）活性。观察ＷＦＢ—Ⅴ型电脑控制的增强型体外反搏机治疗失血性休克后对上述指标的影响。结果表明，犬失血性休克后血浆ＭＤＡ明显升高，全血ＳＯＤ活性显著下降。体外反搏治疗可使升高的ＭＤＡ含量降至正常水平，ＳＯＤ活性明显恢复，从而证明体外反搏治疗失血性休克时，并没有因微循环灌流的改善而带来再灌流损伤，反而能使休克时氧自由基产生减少，为临床应用体外反搏治疗休克提供了理论基础。
The authors replicate hemorrhagic shock model in dogs. The content of malondialdehyde (MDA) in plasma was measured by modified thiobarbituric acid method and the whole blood superoxide dismutase (SOD) activity was measured by photochemical method. To observe the WFB-Ⅴ PC-controlled enhanced external pacing machine in the treatment of hemorrhagic shock on the above indicators. The results showed that after the hemorrhagic shock in dogs, the level of MDA was significantly increased and the activity of SOD in whole blood was significantly decreased. External counterpulsation treatment can reduce elevated MDA levels to normal levels, SOD activity was significantly restored, thus demonstrating that the external counterpulsation in the treatment of hemorrhagic shock, and did not improve perfusion caused by microcirculation perfusion injury, but can make Shock oxygen free radicals generated to reduce the clinical application of EECP shock treatment provides a theoretical basis.