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急性胰腺炎时的炎症是胰舒血管素及胰蛋白酶等消化酶释放到胰周围间隙,并吸收进入血循环引起的。作者假设,由于低血容量、血管扩张引起低血压、胰蛋白酶激活俞肾素为肾素,或这些机制的联合作用,可激活肾素一血管紧张素系统。为此测定并比较正常受试者、除外胰腺炎的急腹症患者以及急性胰腺炎患者急性期与恢复期的活性血浆肾素及无活性血浆肾素值。对象及方法:对象分三组。二组12例,为钠平衡正常、非卧床的健康受试者,年龄20～40岁;11组15例,系除外急性胰腺炎的急腹症患者,年龄24～95岁,平均51岁;皿组14例,系根据病史、体征、血清淀粉酶
Inflammation in acute pancreatitis is caused by the release of digestive enzymes such as pancreatic vasodilator and trypsin into the peripancreas of the pancreas and absorption into the blood circulation. The authors hypothesize that the renin-angiotensin system may be activated due to hypovolemia, vasodilatation-induced hypotension, and renin-activated renin-renin, or a combination of these mechanisms. For this purpose, normal subjects, except acute abdomen with pancreatitis, and active and inactive plasma renin values for acute and convalescent acute pancreatitis patients were measured and compared. Objects and methods: The object is divided into three groups. The second group of 12 patients with normal sodium balance, ambulatory healthy subjects, aged 20 to 40 years old; 11 group of 15 patients, except acute abdomen acute pancreatitis patients, aged 24 to 95 years, mean 51 years; Dish group of 14 cases, based on history, signs, serum amylase