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据Nathanson DA 2013年12月6日(Science,2013,December 5.doi:10.1126/science.1241328.)报道,美国路德维格癌症研究所和加利福尼亚大学的研究者通过研究发现,脑癌细胞可以通过减少靶向药物作用的基因突变,从而对癌症疗法产生抗性,随后在疗法被阻断后重新扩大促进癌症生长的突变效应。
According to a report by Nathanson DA on December 6, 2013 (Science, 2013, December 5.doi: 10.1126 / science.1241328.), Researchers at the Ludwig Institute for Cancer Research and the University of California found that brain cancer cells can By reducing genetic mutations that target drug effects, they become resistant to cancer therapies and then re-expand the mutational effects that promote cancer growth after therapies are blocked.