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心力衰竭(heart failure,简称心衰),是一种复杂的临床综合症状,为各种心脏疾病的终末期表现,是引起心脏病患者死亡的重要原因。心衰的代偿机制在一定程度上有益于心衰患者的血液动力学,而过度的代偿又会对机体产生很多不利的影响。神经体液调节机制错综复杂,至今仍未被完全解释,是目前基础医学及临床医学的研究热点。本篇将就交感神经系统(SNS)、肾素-血管紧张素-醛固酮系统(RAAS)、血管活性物质(ET、NO)、炎性及免疫因素、利钠肽类(ANP、BNP)等神经体液因素与心衰的关系进行综述,力图能够全面系统地反映目前国内外在这一领域的研究现状。
Heart failure (heart failure, referred to as heart failure), is a complex clinical syndrome, for the end of all kinds of heart disease performance, is an important cause of death in patients with heart disease. The compensatory mechanism of heart failure to a certain extent, beneficial to patients with heart failure hemodynamics, and excessive compensation will have a lot of adverse effects on the body. The mechanism of neurohumoral regulation is complex and has not yet been completely explained so far. It is a hot spot in basic medicine and clinical medicine. This article will be on the sympathetic nervous system (SNS), renin - angiotensin - aldosterone system (RAAS), vasoactive substances (ET, NO), inflammatory and immune factors, natriuretic peptide (ANP, BNP) The relationship between body fluid and heart failure is reviewed in an attempt to reflect the current status of research in this area both at home and abroad comprehensively and systematically.