探讨了二羧乙基锗倍半氧化物（Ｇｅ－１３２）对肝枯否细胞免疫及酶活性的影响，二羧乙基锗倍半氧化物在体外实验中明显地对抗四氯化碳对大鼠肝枯否细胞的损害、明显增加花环形成率、吞噬率及吞噬指数，而且显著增加过氧化物小体、过氧化物酶、琥珀酸脱氢酶活性．实验结果表明，四氯化碳可以损伤肝枯否细胞，二羧乙基锗倍半氧化物（Ｇｅ－１３２）可保护和修补四氯化碳的细胞损伤． The effect of dicarboxyethylgermanium sesquioxide (Ge-132) on hepatic cellular immunity and enzyme activity was discussed. In vitro experiments of dicarboxyethylgermanium sesquioxide significantly antagonized the effect of carbon tetrachloride on large Hepatic pupal cell damage, significantly increased the rate of rosette formation, phagocytic rate and phagocytic index, but also significantly increased peroxisome, peroxidase, succinate dehydrogenase activity. The experimental results show that carbon tetrachloride can damage Hepatic Fibroblasts cells, and dicarboxyethyl germanium sesquioxide (Ge-132) can protect and repair CTC cell injury.