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电刺激家兔下丘脑近中线区和静脉注射儿茶酚胺以诱发兔室性心动过速,观察到此类室速有其特有的发生和发展过程。静脉注射阿托品可迅速使室性心动过速转为窦性心律。认为在心律失常的发生中,交感——儿茶酚胺系统主要是促进心室异搏点的自律性活动及/或折返机制的形成;而副交感兴奋则抑制窦性冲动的发生及传导,有利于异搏点成为主导节律。两者互为环节.阿托品打断了迷走环节,恢复並加快窦性心律及房室传导,使之超过异搏心律而中止室速。
Electrical stimulation of the hypothalamus near the midline and intravenous injection of catecholamines to induce ventricular tachycardia in rabbits, such ventricular tachycardia was observed to have its unique occurrence and development. Intravenous atropine can be rapidly converted to ventricular tachycardia sinus rhythm. It is believed that in the occurrence of arrhythmia, the sympathetic-catecholamine system is mainly to promote the autonomic activity and / or the reentry mechanism of ventricular isthmus points; while the parasympathetic excitement inhibits the occurrence and conduction of sinus impulses, Become the dominant rhythm. Atropine interrupted the vagus, restore and accelerate the sinus rhythm and atrioventricular conduction, so that it exceeds the heart rate beating and termination of ventricular tachycardia.