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随着与甲状分腺机能密切相关的维生素D,Ca研究的进展,提出了对继发性甲状旁腺中各种病态的维生素D和Ca代谢异常。 例如:慢性肾功能不全,骨软化症,佝偻病,假性甲旁低等疾患、是以血清钙降低为诱因而发生继发性甲旁亢的。这类病态产生的主要原因是,维生素D摄取不足,或由于肾功能障碍而使活化型维生素D不足,导致肠道钙的吸收量降低。患此症时,随着血清甲状旁腺素(PTH)的升高,并且有血清钙及活化型维生素D即:1.25(OH)_2D的降低。
With the progress of studies on vitamin D and Ca closely related to thyroid gland function, various metabolic abnormalities of vitamin D and Ca in secondary pathological parathyroid glands have been proposed. For example: chronic renal insufficiency, osteomalacia, rickets, hypoparathyroidism and other disorders, is based on the reduction of serum calcium as a secondary cause of hyperparathyroidism. The main reason for such morbidity is insufficient intake of vitamin D, or due to renal dysfunction and lack of activated vitamin D, resulting in decreased intestinal absorption of calcium. With this disease, with the increase of serum parathyroid hormone (PTH), and serum calcium and activated vitamin D, namely: 1.25 (OH) _2D decreased.