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1.Gilbert 病:本病于1901年由 Gilbert 氏描述,是一般称做 Gilbert 征候群或 Gilbert 病的非溶血性黄疸。发病多止于青春期,呈持续多年的慢性或间歇性黄疸。过劳、其它疾患、妊娠等均可诱使黄疸增著,有阳性家族史。有时缺乏黄疸以外的自觉症状;或有疲劳,倦怠及胃肠障碍等。肝脾不大,肝功能检查除血清间接胆红素增加外,余大致正常;尿胆红素试验阴性;胆囊造影正常;血液检查无溶血征象。肝活检可发现轻度脂肪浸润或大致正常。间接胆红素增加的原因尚不明了,一般认为系胆红素摄取和输送障碍所致。
1. Gilbert’s Disease: This disease, described by Gilbert in 1901, is nonhemolytic jaundice, commonly referred to as Gilbert’s syndrome or Gilbert’s disease. More than the onset of puberty, showed persistent chronic or intermittent jaundice for many years. Overwork, other diseases, pregnancy, etc. can induce increased jaundice, a positive family history. Sometimes lack of symptoms other than jaundice; or fatigue, fatigue and gastrointestinal disorders. Liver and spleen is not large, liver function tests in addition to serum indirect bilirubin increased, I generally normal; urine bilirubin test negative; normal gallbladder imaging; blood tests without hemolysis signs. Liver biopsy can be found mild or almost normal infiltration of fat. The reason for the increase of indirect bilirubin is not clear, it is generally believed that the Department of bilirubin intake and transport disorders caused.