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对31例乙型病毒性肝炎外周血淋巴细胞染色体常规法以及31例姊妹染色单体互换(SCE)法观察结果表明:乙型肝炎病毒可致DNA 损伤,造成染色体畸变以及SCE 率升高,尤以慢性活动型为著,其原因可能由遗传因素有关的抑制性T 细胞功能缺陷所致。病毒性肝炎是目前常见传染病之一。据研究,乙型病毒性肝炎表面抗原(HBsAg)有明显的家族集聚倾向,并认为乙型肝炎抗原的分布是和常染色体隐性遗传的规律相符,病毒感染是诱发人类染色体畸变的原因之一。为探索乙型病毒性肝炎与遗传的关系,深入了解其致
The chromosomal routine of peripheral blood lymphocytes from 31 cases of hepatitis B virus and 31 cases of sister chromatid exchange (SCE) method showed that hepatitis B virus could cause DNA damage, resulting in chromosome aberration and SCE rate increase, Especially for chronic active type, the reason may be due to genetic factors related to inhibition of T cell dysfunction caused. Viral hepatitis is one of the common infectious diseases. According to the study, the obvious tendency of familial agglutination of HBsAg is that the distribution of hepatitis B antigen is consistent with the autosomal recessive inheritance, and the viral infection is one of the causes of human chromosome aberration . In order to explore the relationship between hepatitis B and genetic, in-depth understanding of its cause