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作者用经氯醛糖、乌拉坦麻醉，肌肉麻痹，人工呼吸的成年家猫２４只。用０．１～０．５ｍＡ，８０Ｈｚ，０．２ｍ的方波脉冲电刺激下丘脑室旁核（ＰＡ）１２秒（ｎ＝９），引起显著的血压增高反应。２２个最大反应位点多位于ＰＡ的后部背内侧份，其中１１个位点伴心率加快１７个位点伴心率减慢１４个位点心率无变化，且均位于ＰＡ的前部。电解损毁双侧孤束核（ＮＴＳ）尾侧半（ｎ＝５）后，电刺激ＰＡ引起的增压反应和心率改变与损毁前无显著性差异，以上结果表明，ＰＡ是调节心血管活动的重要中抠之一；ＮＴＳ可能不参与电刺激ＰＡ引起的增压反应。
The authors used chloralose, urethane anesthesia, muscle paralysis, artificial respiration of 24 domestic cats. Electrical stimulation of the paraventricular hypothalamic nucleus (PA) for 12 seconds (n = 9) with 0.1-0.5 mA, 80 Hz, 0.2 m square wave pulses produced a significant increase in blood pressure. Twenty-two of the maximal response sites were located in the dorsal and medial parts of the PA. Eleven loci had an increase in heart rate at 17 loci with a decrease in heart rate and no change in heart rate at 14 loci, all of which were located in the anterior portion of PA. Electrolytic damage to the caudal hemicentral nucleus (NTS) cauda equina (n = 5), the PA induced electrical stimulation and heart rate changes were not significantly different from the damage before, the above results show that PA is the regulation of cardiovascular activity One of the important mid-pull; NTS may not participate in the stimulation of PA-induced hyperuricemia.