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输精管阻断术后发生附睾管扩张,附睾上皮吸收与分泌活性增强。附睾间质和管腔表现出明显的炎性反应,支持输精管阻断术后生殖道的改变具有免疫学基础。附睾上皮转运机制、血-附睾屏障通透性及附睾管液流速度改变可能影响精子成熟期间赖以生存的微环境。输精管吻合术可阻止附睾病变的发展,但似不能改善或逆转。这可能部分解释输精管吻合术后精子运动力和活力的低下。附睾形态和生物学其它方面不能恢复正常可能在吻合术后持续不育中起有作用。
Vas deferens occurred after epididymal tube dilatation, epididymal epithelial absorption and secretion of enhanced activity. Epididymal interstitial and lumen showed a significant inflammatory response, support vas deferens postoperative reproductive tract changes with immunological basis. Epididymal epithelial transport mechanism, blood - epididymis barrier permeability and epididymal fluid flow velocity changes may affect sperm maturity depends on the survival of the microenvironment. Vasectomy can prevent the development of epididymal lesions, but does not seem to improve or reverse. This may explain, in part, the low motility and viability of sperm after vasectomy. Other aspects of the epididymis morphology and biology can not return to normal may play a role in the continuous infertility after anastomosis.