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ARDS是一种以肺间质和肺泡毛细血管通透性增加为特征的非心源性肺水肿。尽管20年来监护及支持疗法的进展,但其死亡率仍高于50%。ARDS由各种与肺无关系,但最终导致损害肺气体交换的疾病引起,其主要的病理生理改变为肺泡毛细血管通透性增加,引起间质和肺泡水肿,导致功能残气量减少,肺顺应性降低,结果是动脉氧分压(PaO_2)降低,分流比值及生理死腔增加。治疗的目的是在肺急性创伤后,在修复期间支持肺和扭转这些异常改变,此外在维持气体交换及给氧时,应尽可能矫正感染和心力衰竭等因素。
ARDS is a non-cardiogenic pulmonary edema characterized by increased pulmonary interstitial and alveolar capillary permeability. Despite the progress of guardianship and supportive care over the past 20 years, the mortality rate is still above 50%. ARDS is caused by a variety of diseases that are not related to the lung but ultimately result in damage to the exchange of lung gases whose major pathophysiological changes are increased permeability of alveolar capillaries resulting in interstitial and alveolar edema resulting in a reduction in functional residual volume and pulmonary compliance Sexual decline, the result is lower arterial oxygen pressure (PaO_2), shunt ratio and physiological dead space increase. The aim of treatment is to support the lungs and reverse these abnormal changes during acute lung injury following repair of the acute lung injury. In addition, factors such as infection and heart failure should be corrected to the extent possible while maintaining gas exchange and oxygenation.