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应用细胞外记录的电生理学方法 ,在乌拉坦麻醉的大鼠观察了电损毁双侧杏仁中央核前后脑桥臂旁核味觉神经元对四种基本味觉刺激 (即氯化钠、盐酸、奎宁和蔗糖 )反应的变化。根据对味觉刺激的优势反应 ,2 9个记录的味觉神经元中 ,有 14个NaCl优势、9个HCl优势、3个QH2 SO4 优势和 3个蔗糖优势反应神经元。损毁杏仁中央核明显增强臂旁核味觉神经元对盐酸和硫酸奎宁的反应 (P <0 0 1)。氯化钠优势、盐酸优势和奎宁优势反应神经元对盐酸和硫酸奎宁的反应在电损毁杏仁中央核后也明显增强。在破坏杏仁中央核后 ,臂旁核味觉神经元对氯化钠和硫酸奎宁苦味的分辨能力降低。以上结果提示 ,杏仁中央核在大鼠脑桥水平的味觉编码中发挥重要作用 ,它可能是通过参与对味觉的影响来调节机体的摄食行为
The extracellular recording electrophysiological method was used to observe the effect of four kinds of basic taste stimuli (ie, sodium chloride, hydrochloric acid, quinine and Sucrose) reaction changes. Based on the predominant response to taste stimuli, 14 of the 29 metered taste neurons recorded a merit of 15, a merit of 9, a merit of 3 QH2 SO4 and three predominantly sucrose-responsive neurons. The damaged central almond nuclei obviously enhanced the response of parabrachial taste neurons to quinine hydrochloride and hydrochloric acid (P <0.01). The superiority of sodium chloride, the superiority of hydrochloric acid and the reaction of quinine-dominant reactive neurons to hydrochloric acid and quinine sulfate were also significantly enhanced after electrical damage to the almond central nucleus. After disrupting the central almond nucleus, the parabrachial nucleus of taste neurons declines their ability to resolve the bitter taste of sodium chloride and quinine sulfate. The above results suggest that almond central nucleus plays an important role in the taste-encoding of rat pontine level, which may regulate the feeding behavior of the body by participating in the influence on taste