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以N-(2-巯基丙酰基)-甘氨酸(硫普罗宁,TP)为稳定剂,采用水相法合成了荧光较好的水溶性CdTe量子点(TP-CdTe QDs)。本文通过共振瑞利散射光谱(RRS)、荧光光谱(FL)和紫外光谱(UV-Vis),探讨了TP-CdTe QDs与血红蛋白通过静电引力相互作用的机理.研究发现TP-CdTe QDs与血红蛋白通过静电引力相互作用以后,TP-CdTe QDs荧光猝灭发生猝灭同时荧光光谱发生蓝移,体系的共振瑞利散射光谱强度增大.血红蛋白通过静态猝灭,动态猝灭和光诱导电子转移的方式猝灭TP-CdTeQDs的荧光。同时对体系共振瑞利散射增强的原因进行了讨论。
Fluorescence-sensitive CdTe quantum dots (TP-CdTe QDs) were synthesized by aqueous method using N- (2-mercaptopropionyl) glycine (TP) as stabilizer. In this paper, the mechanism of interaction between TP-CdTe QDs and hemoglobin by electrostatic attraction was investigated by RRS, FL and UV-Vis. It was found that the interaction between TP-CdTe QDs and hemoglobin After the electrostatic attraction interaction, the fluorescence quenching of TP-CdTe QDs quenches and the blue fluorescence shifts, and the intensity of resonance Rayleigh scattering spectra of TP-CdTe QDs increases. The quenching of the hemoglobin by static quenching, dynamic quenching and photoinduced electron transfer Fluorescence of TP-CdTeQDs is extinguished. At the same time, the reasons for the enhanced resonance Rayleigh scattering are discussed.