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为探讨超氧化物歧化酶对实验性骨折愈合的影响，本文检测了实验性骨折前后大鼠体内超氧化物歧化酶活力的变化，并观察了使用外源性超氧化物歧化酶以后，骨折部位骨痂组织细胞的形态学变化。结果表明：骨折后大鼠体内超氧化物歧化酶活力明显低于骨折前（Ｐ＜００５）；外源性超氧化物歧化酶可使早期骨痂内毛细血管含量增加，软骨细胞、成纤维细胞功能活跃，基质钙化迅速、完全，骨性骨痂发生早。认为超氧化物歧化酶有促进骨折愈合的作用。
In order to investigate the effect of superoxide dismutase on experimental fracture healing, this paper examines the changes of superoxide dismutase (SOD) activity in rats before and after experimental fracture, and observes the effect of exogenous superoxide dismutase Morphological changes of callus tissue cells. The results showed that the activity of superoxide dismutase in rats after fracture was significantly lower than that before fracture (P <005). Exogenous superoxide dismutase could increase the content of capillaries in early callus, Fibroblasts are active, matrix calcification is rapid, complete, and bony callus occurs early. Superoxide dismutase is believed to promote fracture healing.