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编辑同志:您好! 我近年来从事止血与血栓方面的研究。在这一领域,有几个方面尚不明确,有待今后进一步研究。现就这几方面借贵刊《读者·作者·编者》栏,询求有关专家的解答。 1.肝素-抗凝血酶Ⅲ(AT-Ⅲ)系统:肝素是临床常用药物。临床使用后,血浆AT-Ⅲ↓,活性正常,因此AT-Ⅲ∶Ag↓使得停药后血液呈暂时性高凝状态。肝素在治疗上与ATⅢ的相互关系如何? 2.蛋白C系统:Protein C inhibitor的缺乏与Ⅴ/Ⅷ联合缺陷症的关系:Protein S-C_4bP参与补体、激肽系统的意义:Protein C-Mφ在释放PAl或PA中的作用等如何?
Editor comrades: Hello! I am engaged in hemostasis and thrombosis in recent years. In this area, there are several aspects that are not yet clear and need further study in the future. Now on these areas by your magazine “readers · author editor” column, ask the experts for their answers. 1. Heparin - antithrombin Ⅲ (AT-Ⅲ) system: Heparin is commonly used in clinical medicine. After clinical use, plasma AT-Ⅲ ↓, normal activity, so AT-Ⅲ: Ag ↓ makes the blood was temporarily hypercoagulable state after stopping. Protein C system: The lack of Protein C inhibitor and Ⅴ / Ⅷ joint defects: Protein S-C_4bP involved in complement, kinin system of significance: Protein C-Mφ in the What is the role of releasing PA1 or PA?