基因失控可以使正常细胞异常生长，形成肿瘤细胞。近年研究发现，染色质末端特异结构端粒（ｔｅｌｏｍｅｒｅ）的不断缩短或丢失，可阻止细胞的增殖；而端粒酶（ｔｅｌｏｍｅｒａｓｅ）的活化，又可以自身ＲＮＡ为模板，在细胞染色质末端不断合成端粒ＤＮＡ序列，维持端粒长度 Gene out of control can make normal cells grow abnormally and form tumor cells. Recent studies have found that the shortening or loss of specific telomeres at the ends of chromatin can prevent the proliferation of cells, while the activation of telomerase can be synthesized from the ends of chromatin at the end of their chromatographic processes using their own RNA. Telomere DNA sequence, maintaining telomere length