Chronic Stress Induced Proliferation and Activation of Mast Cells of Gastric Antrum in Rats

来源 :Wuhan University Journal of Natural Sciences | 被引量 : 0次 | 上传用户:lw3202004012
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To explore chronic heterotypic stressors induced proliferation and activation of mast cells in gastric antrum mucosa by ultrastructure alterations of mast cells,chronic unpredictable heterotypic stressors were used as a study model.The mean immunofluorescence magnitude of mast cell protease 1 (MCP-1) taken from chronic stress rats were 37.4±7.7,significantly higher (p<0.05) than the value of normal control group (24.8±5.6).Chronic unpre-dictable haterotypic stressors appeared also to induce ultrastructure alterations of mast cells.It indicated that mast cells were proliferated and activated,while mast cell granules were hyperplasiaed.Most granules had obvious intragranular changes with loss of electron-dense material.The morphologic evidence showed that macro-phages and leukocytes infiltrated in chronic stress rats.Some granules of leukocytes adhered to the surface of mast cell,and formed a bridge.Macrophages phagocytized the mast cell granules. To explore chronic heterotypic stressors induced proliferation and activation of mast cells in gastric antrum mucosa by ultrastructure alterations of mast cells, chronic unpredictable heterotypic stressors were used as a study model. The mean immunofluorescence magnitude of mast cell protease 1 (MCP-1) taken from Chronic unpre-dictable haterotypic stressors were also indicated by ultrastructural alterations of mast cells. It was indicated that mast cells were 37.4 ± 7.7, significantly higher (p <0.05) than the value of normal control group (24.8 ± 5.6) proliferated and activated, while mast cell granules were obvious intragranular changes with loss of electron-dense material. The morphologic evidence was that macro-phages and leukocytes infiltrated in chronic stress rats. These granules of leukocytes adhered to the surface of mast cell, and formed a bridge. Macrophages phagocytized the mast cell granules.
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