胃食管反流病(GERD)的病理生理机制是食管抗反流屏障功能低下,胃内容物反流到食管引起病人不适症状与食管黏膜损伤的疾病。目前的研究表明,反流物主要为胃酸。胆汁或非酸反流在引起GERD症状的因素中地位有限。由于无法从根本上纠正引起胃食管反流的病理生理机制,目前的治疗措施 The pathophysiological mechanism of gastroesophageal reflux disease (GERD) is the dysfunction of the esophageal anti-reflux barrier, the reflux of gastric contents into the esophagus, and the diseases causing esophageal mucosal damage. The current study shows that reflux mainly stomach acid. Bile or non-acid reflux is limited in the factors that cause GERD symptoms. Because it is impossible to fundamentally correct the pathophysiology of gastroesophageal reflux, the current treatment measures