氧自由基是机体正常代谢的中间产物。心肌细胞缺血损伤时,可产生大量的氧自由基。超氧化物歧化酶(SOD)是氧自由基的清除剂。本文通过测定SOD活性,发现豚鼠于静滴哇巴因中毒出现室早时,SOD活性有明显降低。给予静注刺五加后,SOD活性迅速提高,与对照组比较,有非常显著的差异(P<0.01)。 Oxygen radicals are intermediate products of the body’s normal metabolism. Myocardial cells can produce large amounts of oxygen free radicals when they are damaged by ischemia. Superoxide dismutase (SOD) is a scavenger of oxygen radicals. In this paper, by measuring the activity of SOD, it was found that the activity of SOD in guinea pigs was significantly decreased when guinea pigs developed early ventricular ouabain poisoning. After intravenous administration of Acanthopanax senticosus, SOD activity increased rapidly, and there was a significant difference compared with the control group (P<0.01).