最近,有人探讨用免疫增强剂治疗获得性免疫缺陷综合征(AIDS),也有用免疫抑制剂(如环孢菌素A)进行治疗的,后一疗法的理论基础是,AIDS的早期变化可能是自身免疫。作者发现,活化的T细胞能产生引起AIDS的病毒HTLV-Ⅲ/LAV,而静止的T细胞则不能产生该病毒。因而,设计用抗CD4~+或抗Ⅱ型HLA的单克隆抗体来去除这种能产生病毒的CD4~+细胞,抗CD4~+特异性抗体还能阻止HTLV-Ⅲ/LAV与CD4~+细胞结合。这种方法应尽可能用于AIDS早期。 Recently, some people have discussed the treatment of immunodeficiency syndrome with immune deficiency syndrome (AIDS), but also immunosuppressive agents (such as cyclosporin A) for treatment, the latter therapy is based on the theory that early changes in AIDS may be self-immune. The authors found that activated T cells produce the virus that causes AIDS, HTLV-III / LAV, whereas resting T cells do not produce the virus. Therefore, we designed a monoclonal antibody against CD4 + or anti-HLA class II to remove this virus-producing CD4 ~ + cells, and anti-CD4 ~ + specific antibodies also prevented HTLV-Ⅲ / LAV and CD4 ~ + cells Combined. This method should be used as early as possible in AIDS.