论文部分内容阅读
巴比妥类药巴比妥类药对CNS保护的主要机理是能降低脑代谢率,从而改善氧供/需比值;确切地说这类药物能选择性地降低突触传导耗能同时维持细胞基本功能所需能;它们使CBF向缺血组织再分布且降低颅内压(ICP),也对改善代谢起辅助作用。局灶性缺血巴比妥类药物对局灶性缺血有特殊的保护作用。局灶性缺血时尽管氧供减少,突触活动仍然存在。巴比妥类药选择性降低
The main mechanism by which the barbiturates barbiturates protect CNS is to reduce brain metabolic rate and thereby improve the oxygen supply / demand ratio; rather, these drugs selectively reduce the energy expenditure of synaptic conduction while maintaining the cell Essential functions are required; they redistribute CBF to ischemic tissue and reduce intracranial pressure (ICP), also aiding in improving metabolism. Focal ischemic barbiturates have a specific protective effect on focal ischemia. Although oxygen is reduced during focal ischemia, synaptic activity persists. Barbiturates are selectively reduced