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几周内逐渐形成的亚急性大块肺栓塞的死亡率很高。对标准抗凝血或溶解血栓剂治疗效果不好。其原因可能因栓塞形成较久,其中所含纤维蛋白溶酶原随着栓塞形成时间而逐渐减少,而纤维蛋白溶酶原又是链激酶溶解血块的重要物质,因而疗效不好。据报导外围静脉的陈旧血栓,通过纤维蛋白溶酶原与链激酶治疗;以及急性肺动脉栓塞,应用纤维蛋白溶酶原和尿激酶,都可以使血栓溶解。我们亦有类似情况,报导如下:
Subacute massive pulmonary embolism, which develops gradually in a few weeks, has a high mortality rate. The standard anticoagulant or thrombolytic therapy is not effective. The reason may be due to the formation of embolism for a long time, which contains plasminogen with embolization time gradually decreased, and plasminogen streptokinase is also an important material to dissolve the clot, and thus curative effect is not good. Thrombosis has been reported with obsolete thrombi in peripheral veins, treatment with plasminogen and streptokinase, and acute pulmonary embolism using plasminogen and urokinase. We have similar situations as follows: