Plants have evolved a sophisticated network to fine-tune wound-induced systemic response/resistance(WSR).Jasmonic acid(JA)and its oxylipin relatives,referred as jasmonates(JAs),typi-cally accumulate not only at the injury site but also in distal,unchal-lenged tissues in WSR.In this scenario,certain mobile signal(s)must be translocated from the wounded spot to the undamaged sites to trigger a systemic JA burst by either stimulating de novo JA biosynthesis or direct JA translocation.Both JA-independent and JA-dependent mobile signals in triggering WSR have been proposed to date.The former is represented by the electrical signal mediated by clade three GLUTAMATE RECEPTOR-LIKE(GLR3)proteins,which can be rapidly translocated from leaf-to-leaf in Ara-bidopsis upon wounding,thus provoking JA burst in distal leaves(Mousavi et al.,2013).Nevertheless,the specific JA molecules that act as mobile signals for JA-dependent signal transmission and their mode of action remain elusive.