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针刺力竭肌或延迟性结构损伤肌可促使细胞内增高的Ca~(2+)迅速下降。为探讨针刺的降Ca途径，观察了针刺对肌浆网Ca，Mg—ATP酶、肌膜Ca，Mg—ATP酶和Na，K—ATP酶的影响。研究发现，长时间重复收缩至力竭后、蛙腓肠肌三酶活性分别下降60％、30％、55％。休息24h酶活性仅略有恢复。如力竭后即刻给予肌肉针刺，酶活性恢复加快，然而针后即刻，不论力竭肌还是延迟性结构损伤肌，肌膜、肌浆网Ca，Mg—ATP酶总酶活性均无改变（Ca—ATP酶活性下降，Mg—ATP酶活性上升），但Na，K—ATP酶活性显著上升。结果表明：针刺运动性损伤肌的降Ca作用与肌浆网、肌膜Ca泵主动转运功能无关，但针刺能促进Mg－ATP酶（基酶）活性迅速恢复及肌膜Na—K泵的主动转运。
Acupuncture exhaustive muscle or delayed structural damage can promote intracellular Ca 2+ increased rapidly. In order to explore the mechanism of acupuncture lowering Ca, the effects of acupuncture on sarcoplasmic reticulum Ca, Mg-ATPase, Ca, Mg-ATPase and Na, K-ATPase were observed. The study found that repeated prolonged contraction to exhaustion, frog gastrocnemius enzyme activity decreased by 60%, 30%, 55%. 24h rest activity only a slight recovery. The muscle acupuncture immediately after exhaustion, enzyme activity recovery accelerated, however, immediately after the needle, regardless of the exhaustive muscle or delayed structural damage muscle, sarcolemma, sarcoplasmic reticulum Ca, Mg-ATPase total enzyme activity were not changed Ca-ATPase activity decreased, Mg-ATPase activity increased), but Na, K-ATPase activity increased significantly. The results showed that: the acupuncture exercise injury muscle decreased Ca and sarcoplasmic reticulum, sarcoplasmic Ca pump active transport function has nothing to do, but the acupuncture can promote Mg-ATPase (enzyme activity) rapid recovery and sarcolemma Na-K pump The initiative transfer.