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某些类型的AVM 术后出现严重的脑水肿和出血,这可能与AVM 高流低阻分流导致周围脑组织慢性低灌有关,当突然阻断分流时可致邻近脑组织极度充血;这种现象Spetzler 等称为正常脑灌注压突破。作者对1983～1986年间接受了手术治疗的62例AVM 患者其手术前后的临床和影象学资料进行分析,以确定何种特征与这种充血性并发症(HCs)及不良后果有关。结果:1,充血性并发症(HCs):血管造影示病灶周围低灌发生了HCs 者为35%,而无盗血现象者则为13%(P<0.05)。有穿通动脉供血发生HCs者为53%(10/19),反之则为7%(P<0.001)。供血动脉总直径<8mm 发生HCs 者为7%,8～15mm 者为
Severe cerebral edema and hemorrhage in some types of AVM postoperatively may be related to the low perfusion of AVM resulting in chronic hypoperfusion in the surrounding brain tissue which can result in extreme hyperemia of adjacent brain tissue when the shunt is suddenly blocked Spetzler and other known as normal cerebral perfusion pressure breakthrough. The authors analyzed the preoperative and postoperative clinical and imaging data of 62 AVM patients who underwent surgical treatment between 1983 and 1986 to determine which characteristics were associated with this congestive complications (HCs) and adverse outcomes. Congestive complications (HCs): Angiography showed 35% of HCs with hypoperfusion around the lesion compared with 13% of those with no steal (P <0.05). There were 53% (10/19) of those who had HCS through the artery and 7% on the contrary (P <0.001). The total diameter of blood supply artery <8mm occurred in 7% of HCs, 8 ~ 15mm were