在网状内皮系统功能抑制的基础上,失血性休克可恒定地引起白细胞由肝脾释放,入肺扣押,并有肺系数明显增大和相应的肺结构损伤。这些现象可被隔离灌流肠道循环而全部消除;又可用内毒素补偿而使其完全重新再现。“肝脾释放”与“肺扣押”,“肺扣押”与肺系数增大,均呈明显正相关;而且肺系数增大与肺结构改变也是相符合的。 Based on the inhibition of reticuloendothelial system function, hemorrhagic shock can invariably lead to the release of white blood cells from the liver and spleen, sequestration into the lungs, and a marked increase in pulmonary coefficient and consequential lung structural damage. These phenomena can be isolated and perfused intestinal circulation and eliminate all; can also be used to compensate for endotoxin completely re-reproduction. “Liver and spleen release” and “lung seizure”, “lung seizure” and increased lung coefficient, were significantly positive correlation; and lung coefficient increases and changes in lung structure are also consistent.