近年来,许多实验资料证明急性心肌衰竭可继发于失血性休克[1][2][3]、烧伤性休克[4]、内毒索性休克[5][6][7]、脓毒性休克[8][9],内脏动脉闭塞性休克[10]和过敏性休克[11]。无论哪种类型休克一旦并发急性心肌衰竭,便可导致休克恶化,以致不可逆[12]。 急性心肌衰竭的主要病理生理学变化特点是心脏指数下降、左心室舒末压增高、左心室收缩压下降、 In recent years, many experimental data prove that acute myocardial failure can be secondary to hemorrhagic shock [1] [2] [3], burn shock [4], endotoxic shock [5] [6] [7], septic Shock [8] [9], visceral arterial occlusive shock [10] and anaphylactic shock [11]. No matter what type of shock complicated by acute myocardial failure, can lead to shock deterioration, so irreversible [12]. The main pathophysiological changes of acute myocardial failure is characterized by decreased cardiac index, increased left ventricular end-diastolic pressure, decreased left ventricular systolic pressure,