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锰是毒性很小的微量元素之一,它在成年鼠中的转换直接与从饮食摄入的水平有关。幼鼠由于组织屏障的效能不佳,在接触锰后,其吸收量相对较高。已知锰和铁具有相同的转运机制。缺铁时,铁和锰的吸收量均增加。一些作者也曾观察到,慢性接触锰的幼鼠和缺铁饮食组的鼠,其脑和垂体的锰蓄积量增加。因此,作者进行了下述实验,以评价慢性接触Mn_3O_4伴缺铁对大鼠生植系统发育的影响。用受孕第1天的Lory-Evans大鼠,随机分为8组,4组给予含240 mg/kg铁的饲料,另4组给予含20 mg/kg铁的饲料(均用Fe(NH_4)_2(SO_4)_2、6 H_2O形式)。两种饲料均含
Manganese is one of the less toxic trace elements and its conversion in adult mice is directly related to the level of dietary intake. Due to the poor performance of the tissue barrier in young rats, their uptake is relatively high after exposure to manganese. It is known that manganese and iron have the same transport mechanism. Iron deficiency, the absorption of iron and manganese are increased. Some authors have also observed that manganese accumulation in the brain and pituitary glands increases in rats chronically exposed to manganese and iron deficient diets. Therefore, the authors conducted the following experiments to evaluate the effects of chronic exposure to Mn_3O_4 with iron deficiency on the development of the reproductive system in rats. Lory-Evans rats on the first day of conception were randomly divided into 8 groups, 4 groups were fed with 240 mg / kg of iron, and the other 4 groups were fed with 20 mg / kg of iron (Fe (NH4) 2 (SO_4) _2,6 H_2O form). Both feeds are included