顽固性肝硬化腹腔积液是肝硬化严重,肝功能和门脉系代偿功能障碍的标志[1,2]。在全部肝硬化腹腔积液中约占16.1%,临床治疗难度较大,预后发展不良,常常迁延不愈最终因出现多种并发症或多脏器损害而死亡。近年来研究认为:肝硬化腹腔积液形成的主要机制是由于有效循环血容量与肾灌注不足引起肾钠、水潴留所致[3]。我院2006年9月—2010年6月对以限钠、限水、利尿等常规治疗无效的 Intractable cirrhosis of the peritoneal effusion is a sign of severe cirrhosis, liver function and compensatory dysfunction of the portal system [1,2]. In all cirrhosis of the peritoneal effusion, accounting for about 16.1%, clinical treatment more difficult, poor prognosis, often delayed unhealed eventually due to a variety of complications or multiple organ damage and death. In recent years, studies suggest that: the main mechanism of the formation of cirrhotic ascites is due to the effective circulating blood volume and renal insufficiency caused by renal sodium, water retention caused [3]. Our hospital from September 2006 to June 2010 to limit sodium, water, diuretic and other conventional treatment is invalid