患者,男,35岁,于1991年3月2日因纠纷自服氧乐果乳剂约30ml,半小时后被发现急诊入院.检查:患者呈浅昏迷,全身出汗,四肢肌肉震颤,双肺闻及大量湿鸣,心率120次/min,心音低钝;双侧瞳孔缩小如针尖状.光反射消失,眼底窥不进.诊断为急性氧乐果中毒,重度,收住内科治疗.经反复洗胃,大剂量阿托品、皮质类固醇及对症处理,10天后全身中毒症状消除,痊愈出院.出院后10天.突觉双目视物模糊,未予重视.次日症状明显加重,双目失明伴头痛、呕吐、眼球深部疼痛,再次入院.检查:全身情况如常.视力:双眼眼前数指,眼球压痛明显,瞳孔散大约6mm,对称,光反射迟钝,眼底见双侧视乳头轻度充血,边界略模糊,黄斑中心光反射存在,眼压:右5.5/5.5=2.13kPa,左5.5/6=1.95kPa.(1kPa=7.5mmHg).诊断:球后视神经炎,系有机磷农药氧乐果中毒引起迟发性视神经损害.继续给予口服阿托品0.3mg,每日3次,全身 The patient, male, 35 years old, was found to have an emergency room about half an hour after he was convicted of an omeprazole emulsion of about 30 ml on March 2, 1991. Examination: The patient presented with shallow coma, generalized sweat, limb muscle tremor, Smell and a large number of wet Ming, heart rate 120 beats / min, heart sound low blunt; bilateral miosis diminished, such as pin-shaped reflex disappeared, the eye glimpse into. Diagnosis of acute omethoate poisoning, severe, receiving medical treatment. Gastric lavage, high-dose atropine, corticosteroids and symptomatic treatment, 10 days after the elimination of symptoms of systemic poisoning, discharged from hospital 10 days after discharge .But suddenly binocular vision is blurred, the attention was not obvious. Visual acuity: The number of eyes before the index finger, eyeball tenderness, mydriasis about 6mm, symmetrical, slow light reflex, the fundus see bilateral nipple hyperemia, border Slightly blurred, there is light reflection of the macular center, intraocular pressure: right 5.5 / 5.5 = 2.13kPa, left 5.5 / 6 = 1.95kPa (1kPa = 7.5mmHg). Diagnosis: retrobulbar neuritis, Department of organophosphorus pesticides Omethoate Cause delayed optic nerve damage. Continue to give oral atropine 0.3mg, 3 times a day, the whole body