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采用双抗体夹心ELISA法测定31例肺炎支原体肺炎(初发)患儿,8例随访患儿,41例支气管哮喘发作期对照患儿和30例正常对照组可溶性细胞间粘附分子—1(sICAM-1)浓度。结果表明:肺炎支原体肺炎初发组及随访组血浆sICAM-1较正常对照组明显增高,随访组较初发组低,但无显著统计学意义,肺炎支原体肺炎初发组较支气管哮喘对照组明显增高,但随访组与支气管哮喘对照组比较无显著差别。结果提示:sICAM-1参与了肺炎支原体肺炎的发病过程,可能也是介 导肺炎支原体肺炎诱发或并发支气管哮喘的重要细胞因子之一。
Thirty-one children with mycoplasma pneumoniae pneumonia (initial onset), 8 children with follow-up, 41 children with bronchial asthma attack and 30 normal controls were included in this study. The levels of soluble intercellular adhesion molecule-1 -1) concentration. The results showed that plasma sICAM-1 in primary and follow-up patients with mycoplasma pneumoniae pneumonia was significantly higher than that in normal control group, but no significant difference was found in follow-up group compared with primary one. However, the incidence of primary mycoplasma pneumoniae pneumonia group was significantly higher than that in bronchial asthma control group But there was no significant difference between the follow-up group and the bronchial asthma control group. The results suggest that sICAM-1 participates in the pathogenesis of Mycoplasma pneumoniae pneumonia and may also be one of the important cytokines that mediate or induce bronchial asthma due to Mycoplasma pneumoniae pneumonia.