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采用禁水和水负荷试验对一例高钠血症伴渴感减退患者进行临床研究,观察血钠、血尿渗透压等变化。显示其仍有调节ADH释放机制,但其阈值增高,符合原发性高钠血症。口服双氢克尿噻和醋酸去氨加压素可部分改善上述异常。
A case of hypernatremia with thirst reduction in patients with clinical trials of water and water load test, observed changes in serum sodium, blood and urine osmolality. Showing that it still has mechanisms for regulating ADH release, but its threshold is elevated, consistent with primary hypernatremia. Oral hydrochlorothiazide and desmopressin acetate may partially improve the above abnormalities.