论文部分内容阅读
目的探讨一氧化碳中毒后迟发性脑病的发病机制及影响预后的相关因素。方法回顾性分析96例急性一氧化碳中毒后迟发性脑病患者的临床资料。结果所有患者急性中毒后,均有5～60d的假愈期,之后出现以痴呆、震颤麻痹和精神症状为主的脑病表现,经高压氧、改善脑循环、营养神经等治疗,有效率为84.4%,治愈率34.4%。结论本病的预后与患者年龄、昏迷时间、假愈期、急性期高压氧治疗次数有关。急性一氧化碳中毒后高压氧治疗的时间越早、疗程越长,预后越好。
Objective To investigate the pathogenesis of delayed encephalopathy after carbon monoxide poisoning and the related factors that affect the prognosis. Methods The clinical data of 96 patients with delayed encephalopathy after acute carbon monoxide poisoning were retrospectively analyzed. Results After acute poisoning, all patients had a false recovery period of 5 ~ 60 days, followed by dementia, paralysis and psychiatric symptoms of encephalopathy. After hyperbaric oxygen therapy, cerebral circulation and nutritional nerves were improved, the effective rate was 84.4 %, Cure rate 34.4%. Conclusions The prognosis of this disease is related to patient’s age, coma time, false leave and acute hyperbaric oxygen therapy. Acute carbon monoxide poisoning after hyperbaric oxygen treatment earlier, the longer course of treatment, the better the prognosis.