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骨质疏松症组织学变化表现为骨皮质变薄,中央管(哈氏管)扩大,骨小梁减少,骨小梁间隙增宽,由此而引起疼痛、压缩、变形和易于骨折等一系列功能障碍,称骨质疏松症。原发性骨质疏松的病因尚未完全明了。笔者对目前所知的致病因素和防治,加以综述。1 病因1.1 废用性骨质疏松 废用性骨萎缩可见于长期全身或局部活动减少,肢体瘫痪,慢性骨关节病或因骨折长期固定的肢体;成长中的儿童及成人,因某些疾病引起急性瘫痪(如脊髓灰质炎),使大量的骨形成正在进行,但当骨形成突然中止时,即可出现所谓急性废用性骨质疏松。
Osteoporosis histological changes showed cortical thinning, the central tube (Hastelloy tube) to expand, reduce trabecular, trabecular widening, resulting in pain, compression, deformation and easy fracture and a series of Dysfunction, called osteoporosis. The etiology of primary osteoporosis is not yet fully understood. The author of the currently known risk factors and prevention and treatment, to be reviewed. 1 Etiology 1.1 Disuse osteoporosis Disuseable bone atrophy can be seen in long-term systemic or local activity decreased, limb paralysis, chronic osteoarthritis or fractures due to long-term fixed limbs; growing children and adults due to certain diseases Acute paralysis (such as poliomyelitis) causes a large amount of bone formation to take place, but so-called acute disabling osteoporosis occurs when the bone formation abruptly ceases.