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有人提出,同型半胱氨酸可能通过产生活性氧而诱发内皮功能失调及动脉粥样硬化。曾有报道,同型半胱氨酸促使线粒体损伤。考虑到氧化应激对线粒体生物合成会产生影响,我们提出这样一个假设:在内皮细胞同型半胱氨酸诱发的活性氧导致线粒体生物合成加强。我们发现同型半胱氨酸诱发?
It has been suggested that homocysteine may induce endothelial dysfunction and atherosclerosis by generating reactive oxygen species. It has been reported that homocysteine promotes mitochondrial damage. Given that oxidative stress can have an impact on mitochondrial biosynthesis, we hypothesized that homocysteine-induced ROS in endothelial cells leads to enhanced mitochondrial biosynthesis. We found that homocysteine induced?