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特发性Q-T间期延长综合征是复极延长为尖端扭转型心动过速是一种室性快速心律失常,可引起昏厥或死亡。心电图出现Q-T间隙延长,室性心律失常。其由电解质平衡紊乱,服用影响心室复极的药物等。还可能伴有先天性耳聋,其中最常见的是JerveII-Lange-NeiL-son综合征(常染色体隐性遗传性耳聋)、Romano-Wavcl综合征(常染色体显性遗传性耳聋)。患者有明显Q-T间期延长和形态异常,有发生尖端扭转型室性心动过速的危险并可致死亡。发病时常用β受体阻滞剂首选恢复左右星状神经结的平衡,对心跳暂停依赖性治疗以提高心率为主。除纠正病因外,对症治疗用异丙肾、阿托品或起搏治疗。而禁用Ia、Ic或Ⅲ类心律失常药物。
Idiopathic Q-T prolongation Syndrome is a repolarization extension to torsades de pointes Tachycardia is a ventricular tachyarrhythmia that can cause fainting or death. ECG appears Q-T gap extension, ventricular arrhythmia. The electrolyte imbalance, taking drugs that affect ventricular repolarization. May also be associated with congenital deafness, the most common being JerveII-Lange-NeiL-son syndrome (autosomal recessive deafness), Romano-Wavcl syndrome (autosomal dominant deafness). Patients with significant Q-T interval prolongation and morphological abnormalities, there is the risk of torsades de pointes ventricular tachycardia and can cause death. The incidence of β-blockers often used to restore the preferred choice of the balance of stellate neurons, the heartbeat-dependent treatment to improve heart rate-based. In addition to correcting the cause, symptomatic treatment with isoproterenol, atropine or pacing therapy. While Ia, Ic or type III arrhythmias are disabled.