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为探讨呼吸道病毒感染与气道炎症的关系及喘康平合剂对它的影响 ,将卵蛋白致敏后的豚鼠随机分为模型组、对照组、喘康平组、强的松组和麻杏组。除对照组外各组根据实验需要经呼吸道感染副流感病毒 3型 (P -3) ,观察血浆中P物质 (SP)含量、支气管肺组织病理学改变及肺组织病毒的含量。结果 :致敏豚鼠感染P -3后 ,血浆中SP含量增加 (P <0 0 1) ;光镜下可见支气管上皮脱落、炎性细胞浸润等病理变化 ;喘康平组肺组织病理改变不明显 ;喘康平组和麻杏组P 3含量均明显低于对照组 ,且喘康平组低于麻杏组。说明P -3感染可引起豚鼠的气道炎症 ,喘康平合剂对P -3感染引起的豚鼠气道炎症有保护作用
In order to investigate the relationship between respiratory viral infection and airway inflammation and the influence of chuankangping mixture on it, guinea pigs sensitized with egg protein were randomly divided into model group, control group, chuankangping group, prednisone group, and maxing group. Except for the control group, each group was tested for respiratory infections with parainfluenza virus type 3 (P -3) according to the experimental requirements. The content of substance P (SP) in plasma, pathological changes in bronchopulmonary tissue, and virus content in lung tissue were observed. RESULTS: After sensitized guinea pigs were infected with P-3, the content of SP in plasma increased (P <0 01). Pathological changes such as bronchial epithelium shedding and inflammatory cell infiltration were observed under light microscope. The histopathological changes of lung tissues were not obvious under the chuankangping group. The contents of P 3 in the Chuankangping group and Maxing group were significantly lower than those in the control group, and the Xiekangping group was lower than the Ma Xing group. It is suggested that P-3 infection can cause airway inflammation in guinea pigs, and chuankangping mixture can protect against airway inflammation caused by P-3 infection in guinea pigs