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目的探讨少弱精子症患者生殖道感染与精子质量及精浆中性α-葡萄糖苷酶的相关性。方法分析2011年1月-2015年12月检测的精液分析结果和精浆生化资料。结果少弱精子症的确诊感染者较精浆弹性硬蛋白酶正常者的精液量、精子总数、精浆中性α-葡萄糖苷酶降低,差异有统计学意义(P<0.05)。少弱精子症患者精浆中性α-葡萄糖苷酶测定值随精液量的减少而降低,差异均有统计学意义(P<0.05)。非严重少弱精子症确诊感染组患者的血清FSH测定值高于正常组,差异有统计学意义(P<0.05)。结论生殖道感染可引起少弱精子症患者精浆中性α-葡萄糖苷酶异常。生殖道感染可能是导致少弱精子症患者精液量减少,精子总数下降的重要病因之一。生殖道感染严重可能会影响睾丸生精功能。
Objective To investigate the relationship between genital tract infection and sperm quality and neutral a-glucosidase in seminal plasma in patients with oligoasthenospermia. Methods Semen analysis and seminal plasma biochemical data from January 2011 to December 2015 were analyzed. Results The semen volume, the total number of sperm and the seminal plasma alpha-glucosidase were lower in patients with less sever and less severe spermatozoa than those with normal seminal elastase, the difference was statistically significant (P <0.05). The seminal plasma neutral α-glucosidase in patients with oligoasthenospermia decreased with the decrease of semen volume, the differences were statistically significant (P <0.05). Serum FSH levels in patients with non-severe oligoasthenospermia confirmed by infection were significantly higher than those in normal controls (P <0.05). Conclusion Reproductive tract infections can cause seminal plasma neutral α-glucosidase abnormalities in patients with oligoasthenospermia. Reproductive tract infection may be caused by oligoasthenospermia in patients with reduced sperm volume, the decline in the total number of sperm one of the important causes. Serious reproductive tract infections may affect testicular spermatogenesis.