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为明确青春221中抗叶锈病基因的有效性及其遗传机制,以青春221为父本,分别与感病品种铭贤169和Thatcher以及小麦抗叶锈病近等基因系Lr1、Lr3和Lr23的载体品系杂交,获得F1、F2和F3代群体后,分别在苗期和成株期进行抗病性测定。苗期,青春221对致病类型FGJ/QP、SHJ/GN和PHT/RP的抗病性分别由显性抗叶锈病基因Lr1、Lr3和Lr23控制,对致病类型BGD/HL的抗病性则由上述3对显性抗病基因独立或重叠作用控制,与Thatcher杂交后,青春221中的Lr23趋于隐性遗传并被Thatcher中的1对显性抑制基因抑制;成株期,青春221对优势致病类型PHT/RP和THT/TP混合菌种的抗病性由显性抗叶锈病基因Lr23控制,而Lr1和Lr3均不起作用。
In order to clarify the effectiveness and genetic mechanism of leaf rust resistance genes in young221, the young 221 was used as the male parent to breed the susceptible genes of Lil, Lr3 and Lr23 After the F1, F2 and F3 generations were obtained, the disease resistance was measured at seedling and adult stage respectively. The disease resistance of pathogenicity type FGJ / QP, SHJ / GN and PHT / RP was controlled by the dominant leaf rust resistance genes Lr1, Lr3 and Lr23 at seedling stage and youth 221, respectively. The resistance to pathogenicity type BGD / HL Was controlled by the above three pairs of dominant resistance genes independently or overlappingly. After crossed with Thatcher, Lr23 in recessive 221 tended to recessive inheritance and was inhibited by one pair of dominant inhibitory genes in thatcher; adult stage, youth 221 The disease resistance of the dominant pathogenic types PHT / RP and THT / TP mixed strains was controlled by the dominant leaf rust resistance gene Lr23, while neither Lr1 nor Lr3 was effective.