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本文采用多管微电极胞外记录技术观察了短纯音引起兔内膝体（MGB）神经元的声反应及刺激杏仁体对声反应的影响，并在此基础上观察电泳GANA及其桔抗剂Biculline的效应。实验结果表明：GABA可以抑制MGB神经元的声反应及自发放电活动，而GABAA拮抗剂Bicuculline的作用则相反；电泳GABA对MGB神经元产生同刺激杏仁体一样的抑制效应，并且这种影响可被Bicuculline翻转；嗅鼻沟后缘听区皮层（auditorycortexbehindtherhinalsulcus，ACBRS）区对MGB神经元的下行抑制也可由于电泳Bicuculline而消失，但甘氨酸拮抗剂Strychnine则无任何影响。由此推断：介导杏仁体抑制MGB神经元声反应的最终递质是GABA。
In this paper, the multi-tube microelectrode extracellular recording technique was used to observe the short tone pure tone caused by the rabbit knee (MGB) neurons of the acoustic response and stimulate the amygdala on the acoustic response, and on this basis, the electrophoretic GANA and its orange antagonist Biculline effect. The results showed that: GABA can inhibit the acoustic response and spontaneous firing activity of MGB neurons, while GABAA antagonist Bicuculline has the opposite effect; GABA by electrophoresis produces the same inhibitory effect on MGB neurons as stimulating amygdala, and this effect can be Bicuculline reversed. Downstream inhibition of MGB neurons in the auditory cortexbehindtherhinals ulcus (auditorycortexbehindtherhinalsulcus, ACBRS) region also disappeared due to electrophoretic Bicuculline, but the glycine antagonist Strychnine had no effect. From this, we concluded that the ultimate neurotransmitter that mediates the acoustic response of amygdala to MGB neurons is GABA.