急性胰腺炎(acute pancreatitis,AP)主要是由于胰酶对胰腺及其周围组织的自我消化,继发一系列的器官的功能障碍。正常情况下除脂肪酶(LPS)、淀粉酶(AM)、核糖核酸酶以活性型存在外,其他均以非活性状态存在。在病理情况下,这些酶及相关介质参与了AP的发生和发展。1胰腺的酶学变 Acute pancreatitis (AP) is secondary to the dysfunction of a series of organs secondary to pancreatic enzymes’ self-digestion of the pancreas and its surrounding tissues. Under normal circumstances in addition to lipase (LPS), amylase (AM), ribonucleases exist in the active type, the others are in an inactive state. Under pathological conditions, these enzymes and related mediators are involved in the occurrence and development of AP. 1 pancreatic enzymes change