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戴××,男,38岁,1987年4月1日无明显诱因出现左胸及背部剧痛,2小时后出现双下肢无力。排尿费力,在当地做腰穿,压力正常,无梗阻,脑脊液常规,生化检查正常。经治疗约一个月基本恢复。1988年6月28日无明显诱因再次出现左胸背剧痛。3小时后出现双下肢活动受限,尿潴留,88年7月2日以横贯性脊髓病收入院。查体:神清,视力正常,眼底视乳头边界清。双眼球活动自如无复视,无眼震。余颅神经无示位体征,颈_6以下深浅感觉消失。双上肢肌张力正常,肌力Ⅳ级,双下肢完全瘫,肌张力减低。肱二、三头肌腱反射对称存在。双侧膝、跟腱反射消失,未引出病理反射,克氏征阴性。尿潴留。血常规无异
Dai × ×, male, 38 years old, April 1, 1987 no obvious incentive left chest and back pain, two hours later, both lower limb weakness. Urination laborious, do waist wear in the local, normal pressure, no obstruction, cerebrospinal fluid routine, biochemical tests normal. After about a month of basic recovery. June 28, 1988 there was no obvious incentive to recurrence of left chest back pain. Three hours later, there was limited activity of both lower extremities and urinary retention. On July 2, 88, a transgene myelopathy was admitted to the hospital. Physical examination: God clear, normal vision, clear nipple border retinal clear. Double eyeball activity without diplopia, no nystagmus. I cranial nerve showed no signs, neck _6 following feeling disappeared. Double upper limb muscle tone normal, muscle strength grade Ⅳ, both lower extremities completely paralyzed, muscle tension decreased. Brachial, triceps tendon reflex symmetry exists. Bilateral knees, Achilles tendon reflex disappeared, did not lead to pathological reflex, Kirschner sign negative. Urinary retention. Blood is no different