论文部分内容阅读
几十年来,脑血液循环障碍被解释为婴儿脑性瘫痪的主要发病机制之一.分娩时窒息引起血管壁渗透性增加、血管破裂以及胎儿发育过程中某一阶段的缺氧是导致脑血液循环障碍的重要原因.但是,这些研究直到七十年代并未取得进展,婴儿脑性瘫痪的发生机制至今尚无统一意见.作者对25例本病(年龄为17天至1岁7个月)死亡患儿的脑进行了研究.肉眼观察发现软脑膜水肿及充血.大多数患儿可见小的新鲜出血,波及蛛网膜下腔、室管膜、大脑镰和小脑幕.部分患儿同时存在陈旧性出血和含铁血黄素沉积.显微镜检查发现
Decades of cerebral blood circulation disorders have been explained as one of the major pathogenesis of cerebral palsy in infants.Asphyxia during labor causes increased permeability of blood vessel walls, vascular rupture and hypoxia at some stage during fetal development leading to cerebral blood circulation However, these studies did not make any progress until the seventies, so far there is no uniform opinion on the mechanism of cerebral palsy in infants.The author of 25 patients (aged 17 days to 1 year and 7 months) died Children’s brain was studied. Macroscopic findings of pial edema and hyperemia. Most children showed little fresh bleeding, affecting the subarachnoid space, ependymal, falx and the cerebellar tentorium. Some children also exist old Bleeding and hemosiderin deposition. Microscopic examination revealed