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心室重构是心力衰竭发生发展的分子、细胞学基础,能全面影响患者的心室功能及生存预后。研究发现不同部位心室起搏可通过影响心室不同的激动顺序、压力负荷、心肌血供等因素来促进、延缓或逆转左心室重构(left ventricular remodeling,LVR),进而引起不同的心功能改变。
Ventricular remodeling is the molecular basis of the development of heart failure, cytology can fully affect the patient’s ventricular function and survival prognosis. It has been found that ventricular pacing at different locations can promote, delay or reverse left ventricular remodeling (LVR) by influencing different ventricular activation sequences, stress load and myocardial blood supply, which in turn cause different cardiac function changes.